LiverDiseases

A presentation I gave to AAU team in our teaching. A brief basic overview of alcoholic liver disease and the use of Terlipressin in Variceal bleeds - the cochrane review is used for evidence base.  

HCCUpdate.com – 51yo, recurrent hep C after liver transplantation for HCC, cirrhotic liver disease. Interviews conducted by Neil Love, MD. Produced by Research To Practice.  

Video tutorial on liver failure, outlining acute liver disease including hepatitis, fulminant/decompensated liver disease followed by a discussion on chronic liver disease.  

Video tutorial outlining the basic anatomy and physiology of the liver followed by a summary of acute and chronic liver disease, hepatic failure and cirrhosis.  

https://www.facebook.com/ArmandoHasud... Sorry this has to be two parts, I need a better computer, the movie making program is terrible... https://www.facebook.com/ArmandoHasud...  

https://www.facebook.com/ArmandoHasudungan  

Sorry this has to be two parts, I need a better computer, the movie making program is terrible... https://www.facebook.com/ArmandoHasudungan  

http://www.facebook.com/ArmandoHasudungan  

Understand key differences between the main acute and chronic liver diseases with this clear explanation from Dr. Roger Seheult. Includes discussion of alcoh...  

Causes of hepatomegalyMassive = CRAM Cancer - primary and secondary Right heart failure Alcoholic liver disease with fatty infiltration Myeloproliferative diseases (ie Chronic myeloid laekaemia, PRubraVera,Essential Throbocythaemia) Moderate = FAILL  all of the above plus Fatty liver - from DM, obesity Amyloidosis  

Vitamins A, D, E and K are all stored in the liver, as is vitamin B12. Therefore, in liver damage, you can’t store as many of these as you would like to.   Portal circulation Note that there are no valves in the portal circulation. The portal supply is 70% of the blood that the liver receives. The other 30% is supplied by the hepatic artery.   The four main veins that contribute to the portal system are: Splenic vein Superior mesenteric  

This is a very common cause of liver injury. It is caused by excessive alcohol intake. Typically there will be steatosis of the liver. In this pathological change, fat globules begin to accumulate in the cytoplasm of liver cells. this can be pretty harmless, and as a result, is not very specific for predicting if the liver will develop cirrhosis.  

This is unusual as hepatitis viruses go, as it very rarely causes acute infection. People will only become aware of infection when they develop serious liver disease later in life. 80% of those exposed to the virus will develop chronic infection. Spontaneous late clearance of the disease is very rare. Blood transfusion and blood products are a major means of transmission. It is also though that it can be sexually transmitted, but this is not particularly common.  

It behaves a bit like hep C. Up to 2% of blood donors in US have this – but it is not considered pathogenic.  

 

Inheritance: autosomal recessive Pathology:copper incorporation into caeruloplasmin in hepatocytes and and its excretion into bile is impaired so copper is deposited into organs (firstly liver, then basal ganglia) Clinical: - Presents in children with liver failure/jaundice - Presents in young adults with CNS signs e.g. ataxia, tremor, dysarthria  - Eyes: kayser fleischer rings (gold ring around iris)   

• In the family Hepadnaviridae; common name: Hepadnavirus • Known as the smallest DNA virus • Double stranded, circular 42 nm DNA genome;Virion also called Dan…  

• Detected in some HBV infections.;The antigen is found within certain HBsAg particles. • In blood, HDV (delta agent) contains delta-Ag (HDAg) surrounded by an…  

Good news about coffee and liver disease, but make sure it's the right coffee.  

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