Category

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Is 'Teeth-in-a-Day' for you?
<p>Many current advertisements promise patients 'Teeth-in-a-Day' or 'Teeth-in-an-Hour', offering to replace decayed, infected, or missing teeth with implants and even deliver teeth, all on the same day. While this is a possible treatment option, it is not for everyone. There are a number of limitations and the success depends on proper patient selection, diagnosis, and communication between the surgeon and the restoring dentist. This video discusses immediate implants and immediate teeth and when they may be considered and when they should be avoided.</p>
H. Ryan Kazemi, Dmd
over 10 years ago

3
478
Cerebellar Neuroanatomy
Introduction
Examination of the cranial nerves allows one to "view" the brainstem all the way from its rostral to caudal extent. The brainstem can be divided into three levels, the midbrain, the pons and the medulla. The cranial nerves for each of these are: 2 for the midbrain (CN 3 & 4), 4 for the pons (CN 5-8), and 4 for the medulla (CN 9-12).
It is important to remember that cranial nerves never cross (except for one exception, the 4th CN) and clinical findings are always on the same side as the cranial nerve involved.
Cranial nerve findings when combined with long tract findings (corticospinal and somatosensory) are powerful for localizing lesions in the brainstem.
Cranial Nerve 1
Olfaction is the only sensory modality with direct access to cerebral cortex without going through the thalamus. The olfactory tracts project mainly to the uncus of the temporal lobes.
Cranial Nerve 2
This cranial nerve has important localizing value because of its "x" axis course from the eye to the occipital cortex. The pattern of a visual field deficit indicates whether an anatomical lesion is pre- or postchiasmal, optic tract, optic radiation or calcarine cortex.
Cranial Nerve 3 and 4
These cranial nerves give us a view of the midbrain. The 3rd nerve in particular can give important anatomical localization because it exits the midbrain just medial to the cerebral peduncle. The 3rd nerve controls eye adduction (medial rectus), elevation (superior rectus), depression (inferior rectus), elevation of the eyelid (levator palpebrae superioris), and parasympathetics for the pupil.
The 4th CN supplies the superior oblique muscle, which is important to looking down and in (towards the midline).
Pontine Level
Cranial nerves 5, 6, 7, and 8 are located in the pons and give us a view of this level of the brainstem.
Cranial Nerve 6
This cranial nerve innervates the lateral rectus for eye abduction. Remember that cranial nerves 3, 4 and 6 must work in concert for conjugate eye movements; if they don't then diplopia (double vision) results.
The medial longitudinal fasciculus (MLF) connects the 6th nerve nucleus to the 3rd nerve nucleus for conjugate movement.
Major Oculomotor Gaze Systems
Eye movements are controlled by 4 major oculomotor gaze systems, which are tested for on the neurological exam. They are briefly outlined here:
Saccadic (frontal gaze center to PPRF (paramedian pontine reticular formation) for rapid eye movements to bring new objects being viewed on to the fovea.
Smooth Pursuit (parietal-occipital gaze center via cerebellar and vestibular pathways) for eye movements to keep a moving image centered on the fovea.
Vestibulo-ocular (vestibular input) keeps image steady on fovea during head movements.
Vergence (optic pathways to oculomotor nuclei) to keep image on fovea predominantly when the viewed object is moved near (near triad- convergence, accommodation and pupillary constriction)
Cranial Nerve 5
The entry zone for this cranial nerve is at the mid pons with the motor and main sensory (discriminatory touch) nucleus located at the same level. The axons for the descending tract of the 5th nerve (pain and temperature) descend to the level of the upper cervical spinal cord before they synapse with neurons of the nucleus of the descending tract of the 5th nerve. Second order neurons then cross over and ascend to the VPM of the thalamus.
Cranial Nerve 7
This cranial nerve has a motor component for muscles of facial expression (and, don't forget, the strapedius muscle which is important for the acoustic reflex), parasympathetics for tear and salivary glands, and sensory for taste (anterior two-thirds of the tongue).
Central (upper motor neuron-UMN) versus Peripheral (lower motor neuron-LMN) 7th nerve weakness- with a peripheral 7th nerve lesion all of the muscles ipsilateral to the affected nerve will be weak whereas with a "central 7th ", only the muscles of the lower half of the face contralateral to the lesion will be weak because the portion of the 7th nerve nucleus that supplies the upper face receives bilateral corticobulbar (UMN) input.
Cranial Nerve 8
This nerve is a sensory nerve with two divisions- acoustic and vestibular. The acoustic division is tested by checking auditory acuity and with the Rinne and Weber tests.
The vestibular division of this nerve is important for balance. Clinically it be tested with the oculocephalic reflex (Doll's eye maneuver) and oculovestibular reflex (ice water calorics).
Medullary Level
Cranial nerves 9,10,11, and 12 are located in the medulla and have localizing value for lesions in this most caudal part of the brainstem.
Cranial nerves 9 and 10
These two nerves are clinically lumped together. Motor wise, they innervate pharyngeal and laryngeal muscles. Their sensory component is sensation for the pharynx and taste for the posterior one-third of the tongue.
Cranial Nerve 11
This nerve is a motor nerve for the sternocleidomastoid and trapezius muscles. The UMN control for the sternocleidomastoid (SCM) is an exception to the rule of the ipsilateral cerebral hemisphere controls the movement of the contralateral side of the body. Because of the crossing then recrossing of the corticobulbar tracts at the high cervical level, the ipsilateral cerebral hemisphere controls the ipsilateral SCM muscle. This makes sense as far as coordinating head movement with body movement if you think about it (remember that the SCM turns the head to the opposite side). So if I want to work with the left side of my body I would want to turn my head to the left so the right SCM would be activated.
Cranial Nerve 12
The last of the cranial nerves, CN 12 supplies motor innervation for the tongue.
Traps
A 6th nerve palsy may be a "false localizing sign". The reason for this is that it has the longest intracranial route of the cranial nerves, therefore it is the most susceptible to pressure that can occur with any cause of increased intracranial pressure.
Neurologic Exam
almost 10 years ago

7
396
Cranial Nerve Examination - Abnormal
Cranial Nerve 1- Olfaction
This patient has difficulty identifying the smells presented. Loss of smell is anosmia. The most common cause is a cold (as in this patient) or nasal allergies. Other causes include trauma or a meningioma affecting the olfactory tracts. Anosmia is also seen in Kallman syndrome because of agenesis of the olfactory bulbs.
Cranial Nerve 2- Visual acuity
This patientâs visual acuity is being tested with a Rosenbaum chart. First the left eye is tested, then the right eye. He is tested with his glasses on so this represents corrected visual acuity. He has 20/70 vision in the left eye and 20/40 in the right. His decreased visual acuity is from optic nerve damage.
Cranial Nerve II- Visual field
The patient's visual fields are being tested with gross confrontation. A right sided visual field deficit for both eyes is shown. This is a right hemianopia from a lesion behind the optic chiasm involving the left optic tract, radiation or striate cortex.
Cranial Nerve II- Fundoscopy
The first photograph is of a fundus showing papilledema. The findings of papilledema include
1. Loss of venous pulsation
2. Swelling of the optic nerve head so there is loss of the disc margin
3. Venous engorgement
4. Disc hyperemi
5. Loss of the physiologic cup an
6. Flame shaped hemorrhages.
This photograph shows all the signs except the hemorrhages and loss of venous pulsations.
The second photograph shows optic atrophy, which is pallor of the optic disc resulting form damage to the optic nerve from pressure, ischemia, or demyelination.
Images Courtesy Dr. Kathleen Digre, University of Uta
Cranial Nerves 2 & 3- Pupillary Light Refle
The swinging flashlight test is used to show a relative afferent pupillary defect or a Marcus Gunn pupil of the left eye. The left eye has perceived less light stimulus (a defect in the sensory or afferent pathway) then the opposite eye so the pupil dilates with the same light stimulus that caused constriction when the normal eye was stimulated.
Video Courtesy of Dr.Daniel Jacobson, Marshfield Clini
and Dr. Kathleen Digre, University of Uta
Cranial Nerves 3, 4 & 6- Inspection & Ocular Alignmen
This patient with ocular myasthenia gravis has bilateral ptosis, left greater than right. There is also ocular misalignment because of weakness of the eye muscles especially of the left eye. Note the reflection of the light source doesn't fall on the same location of each eyeball.
Video Courtesy of Dr.Daniel Jacobson, Marshfield Clini
and Dr. Kathleen Digre, University of Uta
Cranial Nerves 3, 4 & 6- Versions
• The first patient shown has incomplete abduction of her left eye from a 6th nerve palsy.
• The second patient has a left 3rd nerve palsy resulting in ptosis, dilated pupil, limited adduction, elevation, and depression of the left eye.
Second Video Courtesy of Dr.Daniel Jacobson, Marshfield Clini
and Dr. Kathleen Digre, University of Uta
Cranial Nerves 3, 4 & 6- Duction
Each eye is examined with the other covered (this is called ductions). The patient is unable to adduct either the left or the right eye. If you watch closely you can see nystagmus upon abduction of each eye. When both eyes are tested together (testing versions) you can see the bilateral adduction defect with nystagmus of the abducting eye. This is bilateral internuclear ophthalmoplegia often caused by a demyelinating lesion effecting the MLF bilaterally. The adduction defect occurs because there is disruption of the MLF (internuclear) connections between the abducens nucleus and the lower motor neurons in the oculomotor nucleus that innervate the medial rectus muscle.
Saccades
Smooth Pursui
The patient shown has progressive supranuclear palsy. As part of this disease there is disruption of fixation by square wave jerks and impairment of smooth pursuit movements. Saccadic eye movements are also impaired. Although not shown in this video, vertical saccadic eye movements are usually the initial deficit in this disorder.
Video Courtesy of Dr.Daniel Jacobson, Marshfield Clini
and Dr. Kathleen Digre, University of Utah
Optokinetic Nystagmu
This patient has poor optokinetic nystagmus when the tape is moved to the right or left. The patient lacks the input from the parietal-occipital gaze centers to initiate smooth pursuit movements therefore her visual tracking of the objects on the tape is inconsistent and erratic. Patients who have a lesion of the parietal-occipital gaze center will have absent optokinetic nystagmus when the tape is moved toward the side of the lesion.
Vestibulo-ocular refle
The vestibulo-ocular reflex should be present in a comatose patient with intact brainstem function. This is called intact "Doll’s eyes" because in the old fashion dolls the eyes were weighted with lead so when the head was turned one way the eyes turned in the opposite direction. Absent "Doll’s eyes" or vestibulo-ocular reflex indicates brainstem dysfunction at the midbrain-pontine level.
Vergenc
Light-near dissociation occurs when the pupils don't react to light but constrict with convergence as part of the near reflex. This is what happens in the Argyll-Robertson pupil (usually seen with neurosyphilis) where there is a pretectal lesion affecting the retinomesencephalic afferents controlling the light reflex but sparing the occipitomesencephalic pathways for the near reflex.
Video Courtesy of Dr.Daniel Jacobson, Marshfield Clini
and Dr. Kathleen Digre, University of Uta
Cranial Nerve 5- Sensor
There is a sensory deficit for both light touch and pain on the left side of the face for all divisions of the 5th nerve. Note that the deficit is first recognized just to the left of the midline and not exactly at the midline. Patients with psychogenic sensory loss often identify the sensory change as beginning right at the midline.
Cranial Nerves 5 & 7 - Corneal refle
A patient with an absent corneal reflex either has a CN 5 sensory deficit or a CN 7 motor deficit. The corneal reflex is particularly helpful in assessing brainstem function in the unconscious patient. An absent corneal reflex in this setting would indicate brainstem dysfunction.
Cranial Nerve 5- Motor
• The first patient shown has weakness of the pterygoids and the jaw deviates towards the side of the weakness.
• The second patient shown has a positive jaw jerk which indicates an upper motor lesion affecting the 5th cranial nerve.
First Video Courtesy of Alejandro Stern, Stern Foundation
Cranial Nerve 7- Motor
• The first patient has weakness of all the muscles of facial expression on the right side of the face indicating a lesion of the facial nucleus or the peripheral 7th nerve.
• The second patient has weakness of the lower half of his left face including the orbicularis oculi muscle but sparing the forehead. This is consistent with a central 7th or upper motor neuron lesion.
Video Courtesy of Alejandro Stern, Stern Foundatio
Cranial Nerve 7- Sensory, Tast
The patient has difficulty correctly identifying taste on the right side of the tongue indicating a lesion of the sensory limb of the 7th nerve.
Cranial Nerve 8- Auditory Acuity, Weber & Rinne Test
This patient has decreased hearing acuity of the right ear. The Weber test lateralizes to the right ear and bone conduction is greater than air conduction on the right. He has a conductive hearing loss.
Cranial Nerve 8- Vestibula
Patients with vestibular disease typically complain of vertigo – the illusion of a spinning movement. Nystagmus is the principle finding in vestibular disease. It is horizontal and torsional with the slow phase of the nystagmus toward the abnormal side in peripheral vestibular nerve disease. Visual fixation can suppress the nystagmus. In central causes of vertigo (located in the brainstem) the nystagmus can be horizontal, upbeat, downbeat, or torsional and is not suppressed by visual fixation.
Cranial Nerve 9 & 10- Moto
When the patient says "ah" there is excessive nasal air escape. The palate elevates more on the left side and the uvula deviates toward the left side because the right side is weak. This patient has a deficit of the right 9th & 10th cranial nerves.
Video Courtesy of Alejandro Stern, Stern Foundatio
Cranial Nerve 9 & 10- Sensory and Motor: Gag Refle
Using a tongue blade, the left side of the patient's palate is touched which results in a gag reflex with the left side of the palate elevating more then the right and the uvula deviating to the left consistent with a right CN 9 & 10 deficit.
Video Courtesy of Alejandro Stern, Stern Foundation
Cranial Nerve 11- Moto
When the patient contracts the muscles of the neck the left sternocleidomastoid muscle is easily seen but the right is absent. Looking at the back of the patient, the left trapezius muscle is outlined and present but the right is atrophic and hard to identify. These findings indicate a lesion of the right 11th cranial nerve.
Video Courtesy of Alejandro Stern, Stern Foundation
Cranial Nerve 12- Moto
Notice the atrophy and fasciculation of the right side of this patient's tongue. The tongue deviates to the right as well because of weakness of the right intrinsic tongue muscles. These findings are present because of a lesion of the right 12th cranial nerve.
Neurologic Exam
almost 10 years ago

17
206
ENT - Mouth Examination.mp4
This video - produced by students at Oxford University Medical School in conjunction with the ENT faculty - demonstrates how to perform an examination of the mouth.
Hussam Rostom
over 8 years ago

4
91
Explaining The Inhaler Technique OSCE Station Guide
Patients with respiratory disease (breathing problems) such as asthma or chronic obstructive pulmonary disease (COPD) often require medication in the form of inhalers. Basically they need to inhale, hence the term inhalers, the medication via their mouth into their lungs.
OSCE Skills
almost 7 years ago

2
63
Different types of Inhaler
There are loads of types of inhalers! You need to be able to tell a patient how to use each type, and what type of drugs can go in each type. With all inhalers, the amount of drug deposited on the mouth and pharynx is very high. Generally it is about 85% (even with good technique). This can improve to about 75% with different types of inhaler. How a spacer works
almostadoctor.com - free medical student revision notes
almost 7 years ago
1
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Time to take periodontitis seriously
Periodontitis is the most common chronic inflammatory disease seen in humans, affecting nearly half of adults in the United Kingdom and 60% of those over 65 years.1 It is a major public health problem, causing tooth loss, disability, masticatory dysfunction, and poor nutritional status.2 Periodontitis also compromises speech, reduces quality of life,3 and is an escalating burden to the healthcare economy. In the UK alone it was estimated to cost £2.8bn (€3.4bn; $4.6bn) in 2008,4 not including raised all cause mortality, an association that has been noted in several populations.5 Worryingly, the disease is often silent, being present for decades before diagnosis and treatment. It can leave a substantial pathological footprint on multiple organ systems, as well as the oral cavity.
bmj.com
almost 7 years ago
1
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Time to take periodontitis seriously | The BMJ
Periodontitis is the most common chronic inflammatory disease seen in humans, affecting nearly half of adults in the United Kingdom and 60% of those over 65 years.1 It is a major public health problem, causing tooth loss, disability, masticatory dysfunction, and poor nutritional status.2 Periodontitis also compromises speech, reduces quality of life,3 and is an escalating burden to the healthcare economy. In the UK alone it was estimated to cost £2.8bn (€3.4bn; $4.6bn) in 2008,4 not including raised all cause mortality, an association that has been noted in several populations.5 Worryingly, the disease is often silent, being present for decades before diagnosis and treatment. It can leave a substantial pathological footprint on multiple organ systems, as well as the oral cavity.
- currently located behind a paywall. Your institution may have access through Athens/Elservier or similar.
bmj.com
almost 7 years ago
1
16
Time to take periodontitis seriously
Periodontitis is the most common chronic inflammatory disease seen in humans, affecting nearly half of adults in the United Kingdom and 60% of those over 65 years.1 It is a major public health problem, causing tooth loss, disability, masticatory dysfunction, and poor nutritional status.2 Periodontitis also compromises speech, reduces quality of life,3 and is an escalating burden to the healthcare economy. In the UK alone it was estimated to cost £2.8bn (€3.4bn; $4.6bn) in 2008,4 not including raised all cause mortality, an association that has been noted in several populations.5 Worryingly, the disease is often silent, being present for decades before diagnosis and treatment. It can leave a substantial pathological footprint on multiple organ systems, as well as the oral cavity.
bmj.com
almost 7 years ago
1
16
Time to take periodontitis seriously
Periodontitis is the most common chronic inflammatory disease seen in humans, affecting nearly half of adults in the United Kingdom and 60% of those over 65 years.1 It is a major public health problem, causing tooth loss, disability, masticatory dysfunction, and poor nutritional status.2 Periodontitis also compromises speech, reduces quality of life,3 and is an escalating burden to the healthcare economy. In the UK alone it was estimated to cost £2.8bn (€3.4bn; $4.6bn) in 2008,4 not including raised all cause mortality, an association that has been noted in several populations.5 Worryingly, the disease is often silent, being present for decades before diagnosis and treatment. It can leave a substantial pathological footprint on multiple organ systems, as well as the oral cavity.
www.bmj.com
almost 7 years ago

1
75

1
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Internal medicine on Instagram: “Systemic AL amyloidosis - macroglossia An enlarged tongue is present in this patient with systemic amyloidosis.”
“Systemic AL amyloidosis - macroglossia
An enlarged tongue is present in this patient with systemic amyloidosis.”
Instagram
over 6 years ago

1
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Internal medicine on Instagram: “Amyloidosis tongue enlargement: Enlarged tongue protruding outside the mouth and showing scalloping of the lateral borders due to teeth…”
“Amyloidosis tongue enlargement:
Enlarged tongue protruding outside the mouth and showing scalloping of the lateral borders due to teeth indentations.”
Instagram
over 6 years ago

1
32
Internal medicine on Instagram: “Severe xerostomia with dry tongue and angular chelitis in Sjögren's syndrome. This patient with Sjögren's syndrome not only has a…”
“Severe xerostomia with dry tongue and angular chelitis in Sjögren's syndrome. This patient with Sjögren's syndrome not only has a profoundly dry tongue,…”
Instagram
over 6 years ago

1
20
Internal medicine on Instagram: “Hereditary haemorrhagic telangiectasia involving the lips”
“Hereditary haemorrhagic telangiectasia involving the lips”
Instagram
over 6 years ago

4
127
Nerve and Blood Supply of the Tongue | Kenhub
This is an article listing all the nerves, arteries and veins responsible for the innervation and blood supply of the tongue. Start learning them here.
kenhub.com
over 6 years ago

1
23

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THE PREMOLAR TEETH
The premolar teeth are transitional teeth located between the
canine and molar teeth. There are two premolars per quadrant and are
identified as first and second premolars. They have at least two
cusps. There is always one large buccal cusp, especially so in the
mandibular first premolar. The lower second premolar may, at times
present with two lingual cusps.
uic.edu
about 6 years ago